Angina Pectoris

Angina Pectoris is ,Ischaemic cardiac Pain of short duration due to lack of sufficient amount of oxygenated blood supply to the cardiac musculáture. (Or, it is a form of ischaemic heart disease; marked by
paroxysmal attacks of substernal pain without evidence of lasting damage to the myocardium).

CAUSES OF ANGINA PECTORIS

Exact cause or aetiology is unknown. Predisposing factors:

Age: middle age.

Sex: male predominate.

  • Habit – Smoking.
  • Food habit – Fatty and hypertensioned personsare more affected.
  • Occluded coronary vessels -(a) syphilitic aortitis;
  • (b) atherosclerosis
  • (c), coronary angitis andembolism.
  • Diminished coronary flow – (a) aortic stenosis; (b) gross aortic incompetence; (c) tight mitral stenosis;(d) severe pulmonary stenosis; (e) high pulmonary
  • Vascular resistance Anoxaemia (a) anaemia; (b) high altitude ncreased work of the heart – (a) hypertension; (b) valvular disease.
  • Precipitating causes -(a) muscular exercise; (b) exposure to cold;(c) heavy meal, etc.
  • Psychogenic factor – mental anxiety.

SYMPTOMS OF ANGINA PECTORIS

  • Angina Is essentially a type of pain thus itself a Symptom.
  • Pain in chest.
  • Location -Mid or upper sternal region; may be felt over the left side of chest.
  • Nature of pain – Classically constricting,_squeczing pressing, or crushing in character, sometime numbing or burning.
  • Duration – usually not more than 2 – 5 minutes. Any cardiac pain of more than 5 min is never due to angina pectoris.
  • Provoking factors usually starts by walking up hill or against the wind, after exertion.weight lifting, emotion, by_ hurrying after meals or heavy meal, by exposure-to cold or by unaccustomed exercise.
  • Pain may_also- be induced by excitement,_anger, fear or apprehension, even during sexualintercourse.
  • Relief – Usually disappears after taking rest for a while or after taking nitroglycerin.

SIGNS

Classical signs are present only during attack.
T
(a) Decubitus – propped up. (b) Attitude – Closed
fist kept over the chest. (c) Face – Deathly pale and anxious. (d) Pulse –
Increase. (e) Respiration – Hurried and shallow. (f) Blood Pressure –
may or may not be raised. (g) Shortly after an attack, tender spots may
be present over the praecordium or along the arms. (Cardiac finding-
usually nil. Heart block

Systemic signs – During attack. . Auscultation = Tick-tack rhythm, S4 and S5 may be audible (gallop rhythm), apex beat may
be shified downwards.

COMPLICATIONS

INVESTIGATION

E.C.G. (may or may not show per ischaemic changes ordinarily) STSegment depression flattening of T wave or sometime elevation of`STsegment.

Selective coronary angiography – In many cases it mayshow definite narrowing of coronary artery in more than two segments.

TYPES OF ANGINA PECTORIS

  • Stable angina This is classical angina pectoris shown due to exercise.
  • Unstable angina Same over it is severe; pain occurs in night, duration of pain 15 minutes or more.
  • Variant angina Periodical waking and waning to time fixation.

PROGNOSIS

Prognosis is worse in the patient who has had multiple cardiac infarcts or who has cardiac failure or who has a family history orcoronary disease. Many men have continued to live for many years by restricting their activities & full expectation of life have been achieved.

MANAGEMENT

Patient should be instructed to lead restful life avoiding mental anxieties,worries, smoking and all other provoking factors.

Physical activities should be minimized.

In obese subjects adequate diet chart should be followed so as to reduce the body weight.

SOMETIMES SURGICAL PROCEDURES MAY BE DONE IN EMERGENCY CASES.

Heart Block

It means recurrent sudden attacks of unconsciousness
caused by impaired conduction of the impulse that regulates the
heartbeat; often applied specifically to atrioventricular heart block.

HEART BLOCK CAUSES

  • Depression of conductivity is most commonly due to ischaemia, fibrosis or inflammation of the A. V. bundle.or to vagal stimulation.
  • Myocardial infarction is the commonest cause.
  • Idiopathic focal fibrosis – is a common cause of chronic heart block.
  • Rheumatic fever and Diphtheria.
  • Drugs, e-g., Digitalis,quinine etc.
  • Cardiomyopathies.
  • Disordered.atrial rhythms, .e.g.auricular fibrillation and flutter.
  • Congenital causes, e.g. ventricular
  • septal defect or maldevelopment of the bundle, etc.
  • Aortic stenosis

TYPES OF HEART BLOCK

  • First Degree – All S.A, impulses are allowed to pass through A,V. node but the time is prolonged.
  • Second Degree – Here S.A. impulses are not allowed to pass through A. V. node from time to time.
  • Third Degree of complete heart block -Here no S. A. impulses are allowed to pass through A. V. node,therefore ventricles generate their own rhythm 30 to 40 per minute.

SYMPTOMS & SIGN

Heart attack is major sign of heart block.

  • First Degree heartblock – can be diagnosed only by E.C.G.
  • Second Degree heart block – here, from time to time, some S. A. impulses are not allowed to pass through A.V.node.
  • Thus auricles contract but not the ventricles resulting in absence of heart sound and missing of pulse.
  • This may occurs at regular Intervals giving rise to 2:1or 3:1 heart blocks or may occur irregularly.E.C.G. helps in the diagnosis.

Third Degree or complete heart block

  • Pulse rate 36 -40 per minute.
  • Rhythm regular; Volume high; Tension – high.
  • Condition of arterial wall may be sclerosed.
  • Character – may be collapsing. The pulse is a fixed pulse.
  • Carotid artery may show carotid dance at a rate of 36 to 40 per minute.
  • Jugular veins – show auricular pulsations at a rate of 72 per minute.
  • Auscultation of mitral area – the intensity time, which is, called canon sound. E.C.G. will be the diagnosis.

COMPLICATIONS

The gravest danger of complete heart block is

Stokes-Adam’s Syndrome

MANAGEMENT

  • Complete Heart Block in acute myocardial infarction requires treatment to prevent undue bradycardia or asystole.
  • Isoprenaline, as anintravenous infusion.
  • Tablet Neoepinine 20-mgm. 1-tablet every 4hourly sublingually.
  • Steroids in some cases may be given.

Note -All the medicine and treatment should be apply only on the direction of your Physician not on the basis of content.

HEART ATTACK (MYOCARDIAL INFRACTION)

Myocardial infarction commonly known as Heart Attack,it occurs when oxygen supply to heart muscles becomes nil, due to occlusion of one of the branches of coronary artery.

CAUSES

  • Coronary Thrombosis
  • Coronary Embolism
  • Sub-intimal haemorrhage
  • Critical lowering of blood pressure
  • Syphilitic aortitis

RISK FACTORS

  • AGE- Commonly After 50 Years.
  • Sex- Mostly Men
  • Hypertension
  • Obesity
  • Stress
  • Smoking ,Alcohol,Unhealthy Diet.
  • Diabetes
  • Gout
  • Burger’s Disease
  • Polycythaemia
  • Hyperthyroidism
  • Brisk Haemorrhage as in – (A)- Peptic ulcer (B)- Surgical Operation

SYMPTOMS

  • Breathlessness ,Restlessness
  • Cough,Sputum,Haemoptysis
  • Shock
  • Severe Sweating
  • Pain- Mild to Severe mostly in the middle of Chest with Left Shoulder and Arm. Jaw and Neck
  • Low Blood Pressure
  • Nausea and Vomiting
  • Pulse -Tachycardia
  • Irregular Heart Beat
  • Cardiac arrest


INVESTIGATION

  • ECG
  • 2D ECHO
  • CRP
  • CBC
  • Troponin I
  • LFT
  • KFT
  • Serum Electrolytes
  • Apoloprotein B (apo B)
  • Lipid Profile
  • Random Blood Sugar, HbA1C
  • NT-proBNP
  • Coronary Angiogram
  • CTCA (CT Coronary Angiogram)
  • Urine Examination

COMPLICATIONS-

  • Sudden Death
  • Heart Failure
  • Pericarditis
  • Ventricular Fibrillation
  • Cardiac Aneurysm
  • Complete Heart Block
  • Thrombo Embolism Pulmonary
  • Cardiac Arrhythmias
  • Neurological Disorders- Coma, Hemiplegia, Syncope, Convulsions.
  • Cyanosis
  • Cardiac arrest

PROGNOSIS-

In About a quarter of all cases of Myocardial Infarction death occurs within the first few minutes without medical care. If there will be no complications within the first four weeks,the Prognosis is favourable.

Sudden death under 40 is not uncommon.Unfavourable features are old age,cardiogenic shock,Cardiac failure,heart block and ventricular arrhythmias.

PREVENTION OF HEART ATTACK-

According to the World Health Organization, as many as 80% of all heart attacks and strokes are preventable. The majority of deaths due to CVD are precipitated by risk factors such as high blood pressure, high cholesterol, obesity, or diabetes, which can, to a large extent, be prevented or controlled through the consumption of a healthy diet, regular exercise and avoiding tobacco. Keeping an eye on your blood pressure, cholesterol levels and blood sugar levels is also very important.

  • Eat healthy
  • Stay Active
  • Lose or manage your weight
  • Get healthy sleep
  • Manage your blood pressure
  • Manage your Blood sugar level
  • Lower your cholesterol
  • Don’t Smoke
  • Avoid Alcohol
  • Manage your stress

MANAGEMENT & TREATMENT-


First Aid

If you think someone is having a heart attack:

If the person is unconscious and unresponsive, does not have a pulse, and an automated external defibrillator (AED) is immediately available – follow the instructions on the AED device.

Have the person sit down, rest, and try to keep calm.

Loosen any tight clothing.

Ask if the person takes any chest pain medicine, such as nitroglycerin for a known heart condition, and help them take it.

If the pain does not go away promptly with rest or within 3 minutes of taking nitroglycerin, call for emergency medical help.

If the person is unconscious and unresponsive, and not breathing or does not have a pulse, call the local emergency number, then begin CPR.

If an infant or child is unconscious and unresponsive, and not breathing or does not have a pulse, perform 1 minute of CPR, then call the local emergency number. 

TREATMENT –

Types of medications

Heart attack treatment involves a variety of drugs. The following list provides an overview of the common types. You can also learn about medication in more detail.

Your health care team will recommend the best combination of medications for your situation.

  • Anticoagulant: Sometimes called blood thinners, these medicines make it harder for clots to form and also keep existing blood clots from getting larger.
  • Antiplatelet agent: Keeps blood clots from forming by preventing blood platelets from sticking together.
  • Angiotensin-converting enzyme (ACE) inhibitor: Relaxes blood vessels and allows them to expand while decreasing resistance by lowering levels of angiotensin II. Allows blood to flow more easily and makes the heart’s work easier or more efficient.
  • Angiotensin II receptor blocker: These medicines inhibit angiotensin II from having effects on the heart and blood vessels. This keeps blood pressure from rising.
  • Angiotensin receptor neprilysin inhibitor: Neprilysin is an enzyme that breaks down natural substances in the body that open narrowed arteries. By inhibiting neprilysin, those natural substances can have their normal effect. That improves artery opening and blood flow, reduces sodium (salt) retention and decreases strain on the heart.
  • Beta blocker: Makes the heart beat slower and with less force, which lowers blood pressure.
  • Combined alpha and beta blocker: Combined alpha and beta blockers may be used as an IV drip for people in hypertensive crisis. They may be prescribed for outpatient high blood pressure use if the person is at risk for heart failure.
  • Calcium channel blocker: Interrupts the movement of calcium into the cells of the heart and blood vessels. May decrease the heart’s pumping strength and relax the blood vessels.
  • Cholesterol-lowering medications: Various medications can lower blood cholesterol levels, but statins are the best first course of action. When statins don’t work, or if a person has serious side effects from statin therapy, other drugs may be recommended.
  • Vasodilator: Relaxes blood vessels and increases the supply of blood and oxygen to the heart while reducing its workload. Available as pills to be swallowed, chewable tablets and as a topical application (cream).

Common medical procedures following a heart attack

You’ll find many common medical procedures for heart attack listed here. For more detailed explanations of these treatments, see our page devoted to cardiac procedure.

  • Angioplasty: Special tubing with an attached deflated balloon is threaded up to the coronary arteries. The balloon is inflated to widen blocked areas where blood flow to the heart muscle has been reduced or cut off.
  • Laser angioplasty: Similar to angioplasty except the catheter has a laser tip that opens the blocked artery.
  • Heart valve surgery: Repairs an abnormal or diseased heart valve or replaces it with a healthy one.
  • Atherectomy: Similar to angioplasty except the catheter has a special tool on its tip to cut away plaque from the artery.
  • Bypass surgery: Treats blocked heart arteries by creating new pathways around the clot so blood can flow to your heart muscle.
  • Minimally invasive heart surgery: An alternative to standard bypass surgery using small incisions.
  • Stent procedure: A stent is a wire mesh tube that is placed during angioplasty to hold open an artery and restore blood flow.
  • Transmyocardial revascularization (TMR): A laser is used to drill a series of holes from the outside of the heart into the heart’s pumping chamber.

In addition to the above treatments, you might hear about implantable medical devices being used to treat certain conditions that increase risk of heart Attack.